【參考文獻】
[1] 中華醫(yī)學會兒科學分會新生兒學組.新生兒缺氧缺血性腦病診斷標準[J].中華兒科雜志,2005,43(8):584.
[2] Ichiba H, Yokoi T, Tamai H, et al. Neurodevelopmental outcome of infants with birth asphyxia treated with magnesium sulfate [J]. Pediatr Int,2006,48(1):70-75.
[3] LeMaire SA, Bhama JK, Schmittling ZC, et al. S100β correlates with neurologic complications after aortic operation using circulatory arrest [J]. Ann Thorac Surg,2001,71(6):1913-1919.
[4] 姜志梅,胡麗娜,李曉捷.膽紅素腦病仔鼠血清肌酸激酶及其腦型同工酶的動態(tài)變化[J].中國臨床康復,2005,9(7):132-133.
[5] Inoue K, Ueno S, Fukuda A. Interaction of neuron-specific K+-Cl- cotransporter, KCC2, with brain-type creatine kinase [J].FEBS Lett,2004,564(1-2):131-135.
[6] 冀亞琦,阮新民,孫培吾,等.體外循環(huán)中S-100 蛋白和神經(jīng)元特異性烯醇化酶的變化及其與炎性細胞因子的關系[J].中國胸心血管外科臨床雜志,2004,11(4):248-251.
[7] 胡央波.新生兒缺氧缺血性腦病血清鎂離子水平的變化及臨床意義[J].現(xiàn)代實用醫(yī)學,2004,16(9):534-535.
[8] Sameshima H, Ikenoue T. Effect of long-term, postasphyxial administration of magnesium sulfate on immunostaining of microtubule-associated protein-2 and activated caspase-3 in 7-day-old rat brain [J]. J Soc Gynecol Investig,2002,9(4):203-209醫(yī)學 全在.線提供m.payment-defi.com.
[9] 湯亞南,趙鳳臨,葉鴻瑁. 新生大鼠缺氧缺血后海馬casepase-3mRNA的表達及硫酸鎂的神經(jīng)保護機制研究[J].中華兒科雜志, 2003,41(3):212-214.
[10] Puka-Sundvall M, Hallin U, Zhu C,et al. NMDA blockade attenuates caspase-3 activation and DNA fragmentation after neonatal hypoxia-ischemia [J]. Neuroreport,2000,11(13):2833-2836.